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Your Eyes Create the Reality You Get in Life???

Discussion in 'Redox Rx' started by Jack Kruse, Jan 11, 2019.

  1. Jack Kruse

    Jack Kruse Administrator

    Do Your Eyes Create the Reality You Get in Life?

    Our eyes and skin are specifically designed to be a full spectrum sunlight bulb. These "bulbs" are powered wireless by sunlight. This is a DC electric light without flicker that comes to us from a condensed form of matter called liquid hydrogen. 125 years ago all we have was kerosene lamps, fire from wood, or Edison incandescent bulbs. Things change quickly in the evolution of man-made light and soon diseases followed.

    Illness is the vengeance of nature for the violation of her laws. It turns out, nature's law around light are the one humans have destroyed most since 1879. That destruction continues right up to the present moment and we ignore it constantly.

    Sunlight is closer to the light from an incandescent bulb, but nothing can be built by man to replace the light of the sun. Why? Edison bulbs are temperature driven light emitters; our eyes are adapted to light from atomic emissions of light made by the processes on the sun. Do you know why this is the case?

    The chaotic mechanism of the photoelectric effect is the short answer. This is one of those laws of nature man has violated. Sunlight has a distinctive topologic effect on the water that surrounds each protein inside of us. This small non-linear effect seems innocuous until you understand the power of it. I have given the public many blogs on this effect in light and how this law more than any other changes us bio-physically using topologic changes in water and in proteins.

    If you want to hear an excellent lecture I gave on this topic go watch the video below as I explain how an order is built from chaos using this law. This video covers the entire topic. The photoelectric effect is built into our sun's light. It has atomic sources and not electric arc discharges to create light.

    The source of incident light creates the speed of electrons that leave surfaces light hits, and that speed of the electrons determines the work that can be done by an electron in our hydrated protein lattice. This is the equation life uses to run our clocks and our time crystal proteins. They tic with every ray of light that falls to us. This light creates the phenomena of time in your life.

    The Photoelectric effect has another law of nature she uses to accomplish the task of living = Fermat's Law. You should look into it.

    TAKE HOME POINT: The light source creates the speed in tissues around these hydrated proteins via Fermat's principle. More speed = More work possible. More Work = More Life = more time.

    Blue light ruins electron speeds on the inner mitochondrial membrane by destroying photoreceptor proteins that use Fermat's law. This slows the speed of the photoelectric actions in us = less work possible = less work = less life = less lifetime = Illness is the vengeance of nature for the violation of her laws. This could even lead to cancer.



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    CITES:

     
  2. Jack Kruse

    Jack Kruse Administrator

    Blue light is an absolute killer for humans because of melanopsin dysfunction. Melanopsin dysfunction is a chaotic topologic defect that occurs in hydrated proteins. Sunlight has a different effect. This photoelectric change is the basis of leptin resistance.

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    Leptin resistance is a photonic process in human biology. So what does blue light and nnEMF lead to give what we've learned about melanopsin/retinal links? It ruins the Bazan effect to ruin the long loop to cause liver level leptin resistance. This blocks DHA to be replaced in cell membranes in the liver and CNS/PNS.

    This causes many communication and memory issues via a broken circadian mechanism via the eye and skin. The pic is about the eye but it was created before you knew melanopsin/retinal was in the subcutaneous fat and skin arterioles. See the pic below = Leptin resistance at liver level = lowered global DHA in liver cell membranes that induce PPARγ-target catalase expression and reduce ROS levels, leading to the inhibition of JAK2/STAT3 = what leptin resistance is inside a cell below the pathway level of Ph.D. or MD understanding.........

    https://www.sciencealert.com/how-blue-wavelengths-light-affect-retinal-cell-tissues-eye-disease
     
    Duchess Sunshine likes this.
  3. Jack Kruse

    Jack Kruse Administrator

    The podcast I just did tonight with Sherrill Sellman on 5G is going to bring the house down when it is released. #whitehot. I did this podcast with her in November 2018. I did two with her this week that were quite different. 1st one was on Adrenal fatigue and light that she wanted to talk about but the one I did today was on 5G and I went all in. It will be electric when released. I unleashed the 5G dragon. I showed where the wizard was located for 5G when the curtain was pulled down. https://whatwomenmustknow.podbean.c...alth-and-healing-is-wrong-with-dr-jack-kruse/
     
  4. Jack Kruse

    Jack Kruse Administrator

    Why is the pupillary response the most critical part of the cranial nerve exam diagnosing TBI
    The ipRGCs are known to connect to many brain regions that regulate very different tasks. The cells tell one part of the brain how bright it is outside so that our pupil can rapidly close—in less than a second. When this does not happen with a TBI patient it alerts us there is a problem with the catecholamines like dopamine or with melanopsin damage in the retina due to the liberation of Vitamin A from this opsin. The same ipRGCs also connect to the master clock in the brain that regulates our sleep-wake cycle. However, it takes several minutes of bright light (blue) to make us fully awake. How the same ipRGCs do these very different tasks with different time scales was not clear until now based upon this study. Some clinicians have realized this for a long time and been using the pupillary light test with blue light and red light to see the damage in the eye clock in TBI cases for years.
    The investigators found that the difference has to do with the way that light detected by the retina reaches the brain. By delivering the mini-SOG to the eyes of the mice, they were able to trace the signal to the part of the brain that constricts the pupil in response to light to identify the pathways.
    It turned out these connections were much stronger—similar to water pouring out of a garden hose. Whereas the connection between the ipRGCs and the master clocks in the SCN in the retina were much weaker—more like drip irrigation. This is because the ipRGCs deliver the light signal to the circadian center in the eye clock phenomena through this slower drip system, it takes longer for any meaningful information to reach and reset the brain clock. It also implies since this system works slower in the retina in TBI’s the use of light has to be altered to reduce the symptoms of photophobia and post-concussive effects.
    CITES:
    https://www.cell.com/cell-reports/fulltext/S2211-1247(19)31183-0
     
  5. Jack Kruse

    Jack Kruse Administrator

    Brain-derived nerve growth factor (BDNF) is markedly altered when ubiquitination is off-kilter. BDNF increases new neuron sprouting and new growth in our brain, but the newly created circuits have to be pruned by the action of melatonin at night, during autophagy. If you have a circadian mismatch, you do not release melatonin properly at night, so you never optimally prune these new arborizations. As a result, your brain does not work well. This especially can affect the paraventricular nucleus (PVN). The PVN is where adrenal fatigue begins. The effects of BDNF are similar to drug addiction in several studies you can read on PubMed. So when you start exercising, the feeling of euphoria is the highest at the beginning, and it goes down from there. BDNF and endorphins are supposed to be yoked by our circadian cycle in their release, but when you have calcium efflux present, they are no longer yoked properly. What protein controls this timing in all eukaryotic cells? Calcium/calmodulin-dependent protein kinase type II. When nnEMF is present it ruins calcium signal in cells at voltage-gated channels. http://neuronline.sfn.org/articles/...-mediated-neuroprotection-in-optic-neuropathy
     
    JanSz and Alex97232 like this.
  6. Randomized Controlled Trial -> Omega-3 effects on BDNF
     
    JanSz likes this.
  7. JanSz

    JanSz Gold

    Question;
    Is it possible that manipulating to have good homocysteine levels will influence DHA/AA ratio positively?

    Another words
    it would be not what you eat, but what you kept
    from what you have put in your mouth.




    https://www.mdpi.com/2072-6643/13/4/1095/htm
    upload_2021-9-9_9-22-32.png

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    https://www.walkinlab.com/products/view/homocysteine-blood-test-plasma

    Note;
    Quest Diagnostic testing is available in New Jersey (and other states where LabCorp is not)


    upload_2021-9-9_9-30-57.png
     
    Last edited: Sep 9, 2021
  8. Surprise, surprise ->
    Serum BDNF Concentrations Show Strong Seasonal Variation and Correlations with the Amount of Ambient Sunlight –
    • The number of sunshine hours (a major trigger to entrain seasonality) in the week of blood withdrawal and the 10 weeks prior to this event positively correlated with serum BDNF concentrations (Pearson’s correlation coefficients ranged: 0.05 – 0.18) and this could partly explain the observed monthly variation. These results provide strong evidence that serum BDNF concentrations systematically vary over the year.
    • The number of sunshine hours per week throughout the year ranged from 2 hours per week to 131 hours per week.
    • The correlations between BDNF concentrations and the number of sunshine hours were largest for the number of sunshine hours in 7 to 8 weeks prior to blood withdrawal. Furthermore, peak serum BDNF values were observed in the early autumn and nadir values in the early spring. So, the seasonality in BDNF expression seems to occur with a time-delay relative to the seasons and their corresponding weather characteristics.
     

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