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WHY SHOULD RESEARCHERS FOCUS ON nnEMF AND LIGHT AND NOT FOOD?

Discussion in 'Redox Rx' started by Jack Kruse, Jan 23, 2019.

  1. Jack Kruse

    Jack Kruse Administrator

    What did Dr. Cunnane miss for the Black Swan in training about AD in this video? He is studying food as the fuel effect to see if this is the mechanism behind it and even his initial results do not show this. He has no idea it is not the FUEL effect, and to his credit, he even says it at the end. The food gurus will misuse this talk because they do not understand the real mechanism is in the cytochrome proteins of the matrix and this becomes critical in how we'll build a full understanding of how altered electromagnetic fields cause AD from light.

    Glucose metabolism drops because cytochrome 1 is destroyed by the electromagnetic environment created by non-solar light sources. This is mediated via melanopsin dysfunction. At the same time, the effect of nnEMF increases AMPk and glucose in the blood (Volkow/Frey).

    This causes a drop in NAD+ and oxygen (Sinclair) and it will affect both glucose and the ability to perform beta-oxidation because of changes in the morphology of the mitochondria at the IMJ. Megan McManus and Dr. Doug Wallace did this work. Ketone metabolism in the human brain will still decline over time as heteroplasmy expands for any mechanistic reason over time either in this alien environment or as they age. The mechanism humans are facing now is the electromagnetic pollution in their environment and this is what drives the fuel effects that Dr. Cunnane lays out. I hope people STOP their myopic focus on the fuel and realize AD is a photoelectric disease tied to light signaling which changes the mitochondrial cytochromes that handle carbohydrates in the matrix in very specific ways.

    https://www.youtube.com/watch?time_continue=19&v=OU26epaihmw
     

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