Parkinson's disease (and AD) begins as an electrical defect.......cause by a quantum topologic deficit

And this is why we are trying to use electrical devices to repair it. The smarter move would be to realize it is is a defect in the photoelectric ability of melanin from tyrosine biophysics.

https://www.medgadget.com/2019/01/i...ain-to-control-parkinsons-other-diseases.html

Using electronics to solve a photonics problem is not a wise path.

 

The fluorescence of Tyr is visible as a pronounced shoulder at 300 nm. The peak of Trp fluorescence in this polypeptide (Bovine PTH) occurs at 347 nm. Position of the peak and the relative quantum yields of the two chromophores, make it possible to detect a distinctive band due to Tyr emission (Edelhoch et al. 1969).

I would assume that any protein containing either tyrosine, tryptophan or phenylalanine would absorb photon energy within the overlapping spectrum of UVA and UVB. It would then stand to reason that any tissue that requires or has within it aromatic amino acids would need ample UVA and UVB in order to function optimally. And, as we know this all starts in the eye coupled with DHA.

 

Dopamine's role as a chemical messenger for light adaptation. Dopamine is released by a unique set of amacrine cells and activates D1 and D2 dopamine receptors distributed throughout the retina. Dopamine is made from an aromatic amino acid which is programmed by UV light. Multiple dopamine-dependent physiological mechanisms result in an increased signal flow through cone circuits and a diminution of signal flow through rod circuits.

Dopamine also has multiple trophic roles in retinal function related to circadian rhythmicity, cell survival, and eye growth. In a reciprocal way, the health of the dopaminergic neurons depends on their receiving light-driven synaptic inputs. Dopamine neurons appear early in development, become functional in advance of the animal's onset of vision and begin to die in aging animals. Some diseases affecting photoreceptor function also diminish day/night differences in dopamine release and turnover. http://www.ncbi.nlm.nih.gov/pubmed/15104164

It should be lost on no Black Swan that dopamine and melatonin are created from AM light by different aromatic amino acids.

Melatonin controls mitochondrial DNA and Dopamine controls how we experience, decipher, and sense time between the inside and the outside world. Creating melatonin also occurs first in the eye is the most critical surface creation for humans. Melatonin is made by the aromatic amino acid tryptophan which absorbs and a very unusual amount of UVC light according to its absorption spectra.

This is light that never reaches the surface of Earth in most places. Dopamine is made from tyrosine, another aromatic amino acid that also absorbs short wave UV light. This is why both molecules are tied to the physiology in the sys and skin and are linked to melanopsin. Melatonin controls all regeneration of the photoreceptors in man except the Muller cells in the eye. Both of these chemical molecules are made by sunlight as it collides with aromatic amino acids in our eye to slow sunlight down so it can become matter. That is how they link to one another.

Now, My Reality #12 blog tells you how they work in a cell.

Most people with Parkinson's disease know that they are deficient in dopamine in the midbrain, but most people do not know that that defect in that area spreads to the region from the eye first in very similar fashion to how a prion works. This is why PD patients have misfolded proteins associated with the disease that mimics how prions operate in disease states. It is a story laid out in the OSF #3 blog post in detail.

 

This is why PD and cataracts have a linkage.

Too much blue light and not enough UV or IR = PD, and cataracts

Cataracts result from the structure of the crystalline proteins that make up the lens in our eyes. Specifically, they form when this structure deteriorates, which causes the proteins to clump together, forming a milky layer over the eye that obstructs vision.

Scientists aren’t entirely sure what causes the proteins to do this (in other words, they’re not entirely certain why cataracts form in the first place).

Black Swans are a bit more sure where it begins because you need light to program the proteins in the eye.

The cornea and lens have proteins that need sunlight programming to keep the proteins from misfolding.

In the eye's lens, there is a unique polar lipid-related to cholesterol that is important in this process called lanosterol.



LOOK AT ALL THOSE PHOTON RINGS IN ^^^^^^^^^^^^^^^^^^^^^IN LANOSTEROL

When will the eye docs connect the dots for you?

 

Lanosterol is a triterpene. Triterpenes are a class of chemical compounds composed of three terpene units with the molecular formula C30H48; they may also be thought of as consisting of six isoprene units. Animals, plants, and fungi all produce triterpenes, including squalene, the precursor to all steroids known in science.

 

Triterpenes exist in a great variety of structures. Nearly 200 different skeletons have been identified. Lanosterol has 4 rings and squalene has none. These skeletons may be broadly divided according to the number of rings present. In general pentacyclic structures (5 rings below) tend to dominate in this family of chemicals that all seem built for solar programming in the cosmos.

 

Cholesterol is part of this family too! This is what cholesterol looks like below. LOOK AT THOSE PHOTON TRAPS!!! When cholesterol is low humans tend to get more cancers too.

 

PD/cataract patients seem to have trouble with LDL cholesterol and Vitamin D levels. Do you want to know why?

Humans have to have 14-Demethylation of lanosterol by CYP51 to eventually yields cholesterol and cholesterol becomes Vitamin D after a photoisomerization step with 312 nm sunlight.

Why did I mention CYP51 here? Do you know what it is?
CYP51 is a Cytochromes P450 protein. CYPs are proteins of the superfamily containing heme as a cofactor and, therefore, are hemeproteins. What did Jack tell you about melanopsin dysfunction in 2018 about blue light and retinal liberation?

Retinal, the aldehyde form of Vitamin A destroys all heme-based chromophores. You starting to see how this all fits yet folks?



This photoisomerization step needs metabolic DDW from the mitochondria to operate at good efficiency. If you're not in the sun outside your redox drops and PD and cataract incidence rise.

BIOPHYSICS 101.

 

Why is there a link between PD/cataracts/pathogens in our blood while Vitamin C is lowered at the same time especially in RBC's?

CYP enzymes have been identified in all kingdoms of life: animals, plants, fungi, protists, bacteria, archaea, and even in viruses. This is why so many astronauts have viral particles in the blood and space is known to foster cataracts and protein folding issues in the eye. However, they are not omnipresent in all bacteria; So space blood infections vary compared to the ones we see on Earth. More than 50,000 distinct CYP proteins are known to man.

Most CYPs require a protein partner to deliver one or more electrons to reduce the iron (and eventually molecular oxygen). Remember electrons must be excited by sunlight to use the photoelectric effect in the photon traps in aromatic amino acids. Based on the nature of the electron transfer proteins, CYPs can be classified into several groups:

• Microsomal P450 systems, in which electrons are transferred from NADPH via cytochrome P450 reductase (variously CPR, POR, or CYPOR). Cytochrome b5 (cyb5) can also contribute reducing power to this system after being reduced by cytochrome b5 reductase (CYB5R).
• Mitochondrial P450 systems, which employ adrenodoxin reductase and adrenodoxin to transfer electrons from NADPH to P450.
• Bacterial P450 systems, which employ a ferredoxin reductase and a ferredoxin to transfer electrons to P450.
• CYB5R/cyb5/P450 systems, in which both electrons required by the CYP come from cytochrome b5.
• FMN/Fd/P450 systems, originally found in Rhodococcus species, in which a FMN-domain-containing reductase is fused to the CYP. (cytochrome 2 in humans is related to this system.
• P450 only systems, which do not require external reducing power. Notable ones include thromboxane synthase (CYP5) for platelets, prostacyclin synthase(CYP8) for PG synthesis, and CYP74A (allene oxide synthase). You should begin to see just how we are beings of light who need solar programming by sunlight and not man-made light.

The most common reaction catalyzed by cytochromes P450 is a monooxygenase reaction, e.g., insertion of one atom of oxygen into the aliphatic position of an organic substrate (RH) while the other oxygen atom is reduced to METABOLIC water which is depleted of deuterium by the cell:

RH + O2 + NADPH + H+ → ROH + H2O + NADP+

BLACK SWAN LEVEL STUFF HERE FOLKS.

So why is PD and Vitamin C linked?

If anyone else bothered to check all the other proteins linked to the mechanism they might learn something deeper about the spectrum of light. And the public might realize why everyone's adrenal stress index is trashed who has PD and cataracts..........= blue light and nnEMF 5G effects.

 

The eye connects directly to the PVN in the brainstem before the dopamine tracts develop in the frontal lobes.........so......what are the implications?

Vitamin C defects happen in the RBC's of the eyes first.

When PVN stress raises we get depleted of endogenous vitamin C. Oral C cannot fix it either because of SVCT1 and SVCT2 I just taught you about in the CPC Patreon series.

Guess what it means for a viral infection like polio that is now in 186 5G cities in 39 states in the USA since we flipped on the 5G switch?
https://cnycentral.com/news/nation-...dren-reaches-186-confirmed-cases-in-39-states

It means we BLACK SWAN DOCS SHOULD EXPECT polio outbreaks to explode acutely in kids where nnEMF stress is now located. Those kids will eventually be a higher risk for cataracts and PD too as they age. Nobody sees what I see because they do not understand where the pieces fit folks.

 

See what happens when you cultivate the ability to think well to connect where the pieces fit.........you see how ignorant people really are. I can fully explain all these weird things in our newly active 5G world while the CDC is impotent to do so.

So who packs your parachute?

This is what we will do in the Kruse Longevity Center.

You are paying me to do the thinking for you as you play catch up to me.

 

So when you read papers that scientists think there is a relationship of PD development to infectious agents you'll know why there is and why they are DEAD wrong that a virus or bacteria cause it.

It is caused by a lack of sunlight or too much ALAN or too much nnEMF that ruins your immune cells to allow the proteins to misfold.

And here is the kicker: This is why there is a linkage of Lyme, Bartonella, and Herpes infections to PD too!!!

LYME, BARTONELLA, VIRAL PARTICLES IN BLOOD = IS A SIGN
It is a sign of a defect in your environment and not inside of you.
Functional medicine and alternative docs are abusing thousands into believing a treatment is needed because they have no idea how solar light programs your adaptive immune system. Save your money on drugs and tests. Change your light hygiene first before you opt for a drug or supplement
WHY? What is the science telegraphing those who think well?

⚕️Does immune response vary by time of day?
Of course, yet researchers/food gurus continue to be "surprised" by the role of circadian rhythm in everything. Light controls the process and not food, pills, or supplements

A 1974 study found that whether a mouse administered E. coli lives or dies depended on the time of day of the injection. Imagine that?
Is this why so many people with Lyme and Bartonella and Viral particles in their blood get confused? When these diseases show up in the blood is the infection real or is it the result of a circadian mismatch in the adaptive immune system which is controlled by lymphocytes activated by solar light based upon what I said above?

Can blue light or nnEMF lead to a false positive diagnosis of Lyme, Bartonella or any other viral infection? Yes, it can. This is why all astronauts and cosmonauts come home with these things in their blood.

So if you have these symptoms in your blood do not be fooled in thinking the defect is in you and floating in your blood. It means your environment is loaded with nnEMF and blue light and the non-toxic pathogens once housed in you are looking to escape an environment that is damaging to your mitochondria.

THAT IS WHAT IT MEANS to the BLACK SWAN in training.
https://medicalxpress.com/news/2017-02-lymphocyte-trafficking-circadian-clock.html

 

Now for more on why 5G and Vitamin C will be linked in all diseases, I expect to explode:

Ketogenic templates affect both SVCT1 and SVCT2 via the oxalate switch to control ascorbate levels to control catecholamines and neurotransmitter health.

This is why humans lost the enzyme to make Vitamin C endogenously because they are not designed to eat like a herbivore as they migrated out of Africa to more stressful climates. It also explains why animals with L haplotypes had to adapt to even higher ketogenic diets to make heat to survive cold.

When you get cold you become ketogenic by design as your light environment varies and you are burning your own fat to make C02 and mitochondrial water and ENDOGENOUS plasma vitamin C from oxalates.

Thiamine (TPP) is also needed for the branched chain ketoacid dehydrogenase complex found in the TCA cycle too. And now you might realize why I wrote the 2018 Patreon posts on the TCA cycle and methionine cycles and shows why seafood/pork is a wise choice in a 5G world! Even the Okinawans knew this!!!


Eating vitamin C does not happen in cold climates because it does not grow in the photosynthetic web of life. It is present in many kinds of seafood. People forget we left our African crib by WATERWAYS. This is why we lost our ability to make Vitamin C. If we live on our own fat as we go we have no need for exogenous Vitamin C. corollary is if our life becomes more then cold stressed we will deplete out endogenous mechanism and as a result, our cortisol and melatonin levels drop like rocks. Today we've built a world that drains our endogenous abilities because we have more light stress. Cold stress and seasonal light stress is all nature gave us to work with as we left Africa.

That is why haplotypes and SNP's and SAP's varies in humans over 200K years. Thanks, Doug Wallace!

All the pieces fit when you see them working in unison in your species. This is why 24/7 ketosis is not needed too in many cases when life is built under the sun. The advice must change when you live in a blue-lit nnEMF 5G city. This is a new reality as the environment varies. If one does this before the advent of blue light or nnEMF oxalate kidney stones were more likely. Today's world makes getting oxalate stones less likely because we are using oxalates as our storehouse for Vitamin C to fix the deficits of the PVN and adrenal stressors we've built.

Few do. Welcome to the submolecular world of the Black swan. I teach people how to think when their environment is changing and varying way below their perceptual level. I am a mitochondria whisperer.

 

Anyone see why I have a SERIOUS disdain for food gurus now? When you believe their bullshit long enough you never get to the truth and you think they were right in calling somebody way wiser then they were CRAZY ten years ago. Who is laughing now paleo/LCHF?



This explains why half truths are killers.............

It is not about diet or exercise.......IT IS ABOUT YOUR LIGHT CHOICES. Game, set, match.

 

I will continue to persist to call out scientists and food gurus who think linearly and harm you. They are not part of this Black Swan dance, and likely never will be with their inability to think. I am exhilarated that they won't be either. I'm basking in my solo fullness as I fluff my feathers calling their nonsense out on social media daily. They embody all the things I reject in this world. They settle for bullshit and I never will. This doctor offers you no apology for pointing out your poor thinking/behavior behind your dietary facades.

 

So if you read all this............this showing up in the media is no shock is it?

https://parkinsonsnewstoday.com/2017/07/14/first-signs-parkinsons-disease-eyes-study/

 

I was following it as you were posting. This is an amazing thread! Great information and a more complete picture of Parkinson's pathophysiology and much more. Thank you.

 

Should it surprise anyone if a neurologist thinks PD is related to infections or sepsis?

Dr. Michael Schlossmacher, the Ottawa neurologist is behind the new Parkinson’s disease theory also pointed out that “there has been a growing body of research suggesting that infection plays a key role in the development of Alzheimer’s disease”.

Now the good doctor has no clue what I know.........but I thought you might like to see things are not far fetched and I am way ahead of where they are must come to solve your problem.

This thread is a view into my mind.

 

His theory is consistent with a growing body of research pointing to a correlation between small intestine bacterial overgrowth (SIBO) and motor fluctuations in Parkinson’s disease.

A study by Fasano et al (2013) demonstrated that “eradication of small intestinal bacterial overgrowth resulted in improvement in motor fluctuations without affecting the pharmacokinetics of levodopa.”
https://www.ncbi.nlm.nih.gov/pubmed/23712625

https://www.ncbi.nlm.nih.gov/pubmed/24637123
These findings are confirmed by a 2014 study from Tan et al demonstrating that SIBO “independently predicted worse motor function” in Parkinson’s disease.

 

When will they wake up?