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Low redox = poor NAD+ recycling at cytochrome 1

Discussion in 'Redox Rx' started by Jack Kruse, Nov 27, 2022.

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  1. Jack Kruse

    Jack Kruse Administrator

    NADH is also the source of the electrons that create oxidative stress. Superoxide formation at Complex I is “steeply dependent” on NADH/NAD+ ratio. Aryl Hydrocarbon Receptor is the master controller of NADH/NAD+ cycling. Bile acid feed back is vital in shutting down the aryl hydrocarbon receptor. This is one reason that removal of the gallv=bladder often causes a fat gain in those with poor NAD+ recycling (circadian mismatch)

    Every disease has a low NAD+/NADH ratio = pseudohypoxia. NAD is an electron carrier that declines with age and is crucial for producing ATP How to increase it?
    - niacinamide (disputed in literature)
    - oxidizing glucose
    - B1 (in foods not pills)
    - magnesium (food not pills)
    - methylene blue
    - pyruvate (in proper feedback)
    - thyroid (tied to the seasonal breakdown of aromatic amino acids)
    - K2 (made by gut bacteria not a pill)
    - lower endotoxin (controlled floral growth)
    - cut out seed oils (non DHA PUFA causes reductive stress)
    StevieJ and John Schumacher like this.
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