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Low Cortisol Levels

Discussion in 'Optimal Labs' started by Eddie Garza, Apr 29, 2015.

  1. JanSz

    JanSz Gold

    upload_2017-2-2_13-16-27.png

    Time 21:41
    transferin saturation <50% preferrable or even <45%
    Ferritin<100 women desirable
    Ferritin<200 men

    Soluble Transferrin Receptor (test)

    ----------
    When phlebotomy, goal
    Ferritin=25
    Hgb=12.5
    then 1-2 month schedule, goal
    Ferritin=50-150
    transferin saturation <45%

    lactoferrin can remove iron 300mg 2x/day
    (apolactoferrin) lef.org


    terapeutic phlebotomy

     
    Last edited: Feb 2, 2017
  2. JanSz

    JanSz Gold

    upload_2017-2-6_10-57-1.png
    .......................
     
  3. JanSz

    JanSz Gold

    The 5ARI Withdrawal Syndrome (5ARI-WS)
    The SilencedAndrogen Receptor (AR) Theory:
    Explaining persistent side effects arising from 5alpha reductase inhibitor (5ARI) use
    By “Awor” and “Mew”, Administrators of Propeciahelp.com (August 2010)
     

    Attached Files:

  4. JanSz

    JanSz Gold

    Selegiline, also known as L-deprenyl, is a substituted phenethylamine.
    At normal clinical doses, it is a selective irreversible MAO-B inhibitor.
    ------------------------------------------------------------------------------------------------------
    @SatoriHeart

    http://www.futurescience.com/deprenyl.html

    © 1991-2014 Jerry Emanuelson

    DEPRENYL


    Deprenyl was developed by Dr. Joseph Knoll of Semmelweis University in Budapest, Hungary during the early 1960's for possible use as an anti-depressant. The medicine initially showed limited usefulness as a treatment for depression; but more than a decade after its initial development, deprenyl was found to be an effective treatment for Parkinson's Disease. (See the note at the end of this chapter, however, about the use of a deprenyl skin patch shown to be effective for depression.)

    In April, 1989, the FDA approved deprenyl for use against Parkinson's Disease, a disease that usually strikes people between the ages of 50 and 70. Parkinson's Disease is an incurable disorder that begins with a characteristic tremor and causes progressive disability, ultimately resulting in death.

    Deprenyl acts on an important chemical in the brain called dopamine. Dopamine is manufactured in the brain from two amino acids that occur naturally in foods: phenylalanine and tyrosine.

    To function properly, the human body requires a multitude of chemicals in well-regulated quantities. For most of these necessary chemicals, the human body has one biological process to manufacture the chemical and another process to break it down. Dopamine is broken down in the brain by a chemical called MAO-B. It is important for good health that the manufacture of dopamine from amino acids and the destruction of dopamine by MAO-B is kept in balance. If the destruction of dopamine by MAO-B occurs at a faster rate than its production from the amino acids, the brain cells that use dopamine will die. The loss of dopamine and the resulting brain damage can cause tremors, rigid muscles, loss of coordination, weakness and death.

    Beginning at about age 45, the destruction of dopamine in the brain by MAO-B begins increasing. The amount of dopamine in the typical about human brain begins decreasing by 13 percent every decade.

    In about 0.5 percent of the population, the decrease in dopamine takes place much more rapidly than the usual 13 percent every ten years. When the dopamine content drops to about 30 percent of normal, these individuals develop the tremors and rigid muscles that are typical of Parkinson's Disease. Parkinson's patients typically experience a decrease in dopamine levels of 30 percent to 90 percent every ten years. Death usually occurs in Parkinson's patients about the time their brain dopamine content falls to 10 percent of normal.

    In the past, the most popular treatment for Parkinson's Disease has been L-Dopa. L-Dopa is an amino acid that is not present in the ordinary human diet. The brain can make dopamine much more easily from L-Dopa than from the tyrosine and phenylalanine usually obtained from the human diet.

    Deprenyl attacks the other end of the dopamine-preservation process by inhibiting the action of the MAO-B.

    During the 1980's, deprenyl, either alone or in combination with L-Dopa, was the most effective known drug treatment for Parkinson's Disease. Researchers complained that few physicians are using deprenyl for Parkinson's Disease. Several months after its approval by the FDA, many physicians still hadn't even heard of the medicine, which is marketed by Somerset Pharmaceuticals under the brand name Eldepryl.

    The Sept. 5, 1990 issue of the Journal of the American Medical Association (JAMA) contained an article titled "Many Researchers, Few Clinicians, Using Drug That May Slow, Even Prevent, Parkinson's." The article re-emphasized the dramatic results of deprenyl as shown in several large-scale human studies. In the JAMA article, Dr. Christopher Goetz, a neurologist, said he is "surprised by how few" physicians are prescribing deprenyl. "They don't understand the implications" of the studies, Goetz said.

    Some of the enthusiasm for using deprenyl in Parkinson's Disease has been dampened after it was discovered that the dosage levels used were often too large, and that determining and maintaining the proper dosage in a clinical setting was more difficult that was first thought.

    Dr. Knoll claims that deprenyl is also effective in preventing Parkinson's Disease. Most other scientists working with deprenyl take this claim seriously but are much more cautious about recommending deprenyl in healthy persons.

    A review article in Clinical Neuropharmacology by Dr. Patricia Sonsalla and Dr. Lawrence Golbe typifies the most prevalent thinking on this subject. Sonsalla and Golbe discuss reasons for believing that deprenyl may be a useful preventive therapy for Parkinson's Disease (PD), warning that in autopsies of elderly persons who had no Parkinson's symptoms during life, eight to 10 percent show pathological indications of Parkinson's Disease.

    Sonsalla and Golbe wrote that "clinically evident PD may then be merely the top of a pathologic iceberg comprising 10 percent of the population over 60 in Western countries. If deprenyl is shown to even modestly delay or attenuate the development of clinical PD, or is widely suspected of doing so, large numbers of people may seek the drug. The still distant prospect of a valid pre-symptomatic test may lend a more rational basis to this practice. In any case, frightened patients and their physicians must understand that there are no data on the safety of deprenyl over a span of several decades."

    According to Dr. Knoll, deprenyl has "proved to be a safe drug in man. Neither hypertensive reactions nor the need for special dietary care were ever encountered during long-term (2-8 years) daily administration of the drug." Knoll said that the lethal dose of the drug is more than 1000 times its effective daily dose. Knoll called this safety margin "remarkable."

    Dr. Knoll also said that deprenyl is an effective treatment for aging. Knoll wrote a lengthy report on his study of the age-retarding effects of deprenyl which was published in a European medical journal in 1989.

    In that study, Dr. Knoll divided 132 male rats of the same age into two groups. One group of 66 rats received an injection of salt solution three times a week. The other group of 66 rats each received a similar injection containing deprenyl three times a week. The treatments began when the rats were 104 weeks-old. The rats that did not receive deprenyl died at an average age of 147 weeks. After 164 weeks, all of the untreated (salt-water injected) rats had died; but all of the deprenyl-treated rats were still alive and healthy. It was not until seven weeks later that the first deprenyl-treated rat died. The last deprenyl-treated rat lived to 226 weeks.

    The average life span of the deprenyl-treated rats was 192 weeks. The researchers considered this to be particularly remarkable since the maximum life span of that strain of laboratory rats is considered to be 182 weeks.

    According to Dr. Knoll, similar life-extending results could be expected in humans. The biological processes deprenyl acts upon to cause its life span extending effects in rats are well-known; and there is general agreement among scientists that deprenyl acts on the same processes in humans.

    Dr. Knoll points to the normal decrease in dopamine in the aging brain as an indication of how deprenyl works to increase life span. He said that it is no coincidence that even the healthiest humans die at about the time the dopamine content of their brain drops below the critical 30 percent level. According to our present knowledge, the neurons (brain cells) that use dopamine are the most rapidly aging neurons in the human brain.



    AGE
    45

    55

    65

    75

    85


    95

    105

    115

    125

    DOPAMINE CONTENT
    100 percent


    87 percent


    74 percent


    61 percent

    48 percent



    35 percent


    22 percent


    11 percent


    0
    Dr. Knoll's advocacy of deprenyl use to extend the human lifespan is controversial. Claims of enhanced longevity in humans without conclusive proof have often caused drug companies to have problems with government regulatory agencies. At one symposium on deprenyl, the organizer of the event, Dr. John Mann, was careful to emphasize that Dr. Knoll's statements about the anti-aging effects of deprenyl have "nothing to do with the claims of any pharmaceutical company."

    If the animal experiments translate directly into the same slowing of the aging process in humans, this would result in a 24 percent increase in the maximum life span of humans along with a stretching of the healthy middle years of human life by 25 to 30 years. Since the natural life span of humans is much longer than laboratory animals, whether deprenyl actually has this effect in humans won't be known for at least another three decades.

    In a report in the August 1992 Journal of the American Geriatric Society, Dr. Knoll concluded his report on deprenyl by saying, "We propose that the healthy population be maintained on 10-15 mg deprenyl weekly starting at age 45 to combat the age-related decline of the nigrostriatal dopaminergic neurons. Prophylactic deprenyl medication seems to offer a reasonable prospect of improving the quality of life in the later decades, delaying the time of natural death and decreasing the susceptibility of age-related neurological diseases."

    The editor of the Journal of the American Geriatric Society appended a note to Dr. Knoll's report stating that "The proposal in this final paragraph is the author's opinion and does not reflect mainstream opinion at the present time." Nevertheless, it is remarkable, and probably unprecedented, for a report in a serious medical journal to propose that all healthy persons above a certain age take a prescription drug on a regular basis.

    Since deprenyl is a prescription medicine, those using it for personal life extension experiments are either scientists or physicians or are persons whom physicians trust to have the knowledge and responsibility to be able to use wisely a medicine such as deprenyl on a long-term basis.

    One of the greatest problems that the healthy individuals using deprenyl face is not a medical problem, but a social one. Deprenyl is broken down in the body to amphetamine and methamphetamine. London researcher G. P. Reynolds and his associates reported in a British medical journal that even in the larger doses used in Parkinson's Disease, deprenyl is "unlikely to produce any marked degree of central amphetamine-like action."

    The amphetamine and the methamphetamine breakdown products of deprenyl can show up in urine, though. Some scientists have warned that this may cause problems for those individuals who must undergo employer required drug testing.

    Methamphetamine is a common illegal "street drug" with the potential for producing addiction. The scientists who work with deprenyl are not concerned that any harm will be caused by the tiny amounts of amphetamine and methamphetamine produced by deprenyl, but concern has been expressed about the dangers to a person's job and reputation from positive results on drug tests.
     
    Last edited: Feb 16, 2017
  5. JanSz

    JanSz Gold

    The Marketing of Deprenyl

    Deprenyl Research Ltd., the company that markets deprenyl in Canada, is researching the use of deprenyl in controlling the symptoms of Alzheimer's disease and in reducing fatigue in multiple sclerosis. The company hopes eventually to secure approval from government regulatory agencies to market deprenyl for these uses. (In mid-1994, Deprenyl Research changed its name to Draxis Health, Inc, reflecting its intention to become involved in other products besides deprenyl.)

    A number of entrepreneurs have been involved in efforts to gain approval to sell deprenyl for extending the lives of dogs, cats and other pets. The first such effort was by Dr. Morton Shulman of Toronto. In 1987, Dr. Shulman was 62 years-old and so severely afflicted with Parkinson's Disease that he was hardly able to move without help. He obtained some deprenyl from Europe after hearing about the drug from a neurologist.

    In Longevity magazine, Dr. Shulman is quoted as saying that "within 24 hours of taking the drug, I stopped shaking and shuffling, returned to normal, and went back to work."

    According to a report in the Oct. 3, 1988 issue of Business Week, after deprenyl halted the course of his disease, Dr. Shulman began stirring up a considerable amount of controversy with his efforts market the drug as a life span extender for pets.

    Shulman made efforts to induce pet food companies to secure official approval for deprenyl for animal lifespan extension from U.S. and Canadian regulators. At that time, deprenyl had yet to be approved by the U.S. FDA for any use; and many people feared that Shulman's claims about life span extension in animals would cause the FDA to slow the approval process in the U.S. This problem was complicated by the fact that Shulman had a financial interest in the company that was applying for FDA approval for human use in Parkinson's Disease.

    According to a report in the Jan. 7, 1991 issue of Barron's, a weekly financial newspaper, a new company, Deprenyl Animal Health, Inc. was formed for the purpose of marketing deprenyl for veterinary use. Barron's quoted company officials as saying that they believe that deprenyl "when chronically administered in low doses, may extend the healthy period of small companion animal's lives by retarding the 'normal' decline of certain physiological functions, particularly in dogs and cats, during the senescent period of such animals lives."

    Deprenyl Animal Health, Inc. later signed agreements with Chinoin of Hungary, the world's primary producer of deprenyl, for exclusive rights to market deprenyl for "veterinary prescription applications" in the U.S. and Canada. In 1992, they established headquarters in Overland Park, Kansas, and began implementing plans to sell a veterinary form of deprenyl under the brand name Anipryl.

    By 1999, television ads for Anipryl, the veterinary form of deprenyl, were commonplace.

    The basic U.S. and Canadian patents on deprenyl that were held by Chinoin of Hungary have expired. However, Chinoin holds patents in both countries on the process used for making deprenyl. Those patents did not expire until Dec. 20, 2003.

    Competing companies were searching for alternative processes for manufacturing the drug that are not covered by the Chinoin patents. One company, Discovery Experimental and Development, Inc., apparently succeeded in using a completely different process to make a liquid form of deprenyl. The company claimed that it was more bioavailable than the Chinoin product. At one time, it was submitted for FDA approval. Subsequently, competitors conspired with rogue agents of the FDA to put Discovery out of business.



    Dangers of Deprenyl

    In spite of the impressive safety record of deprenyl, any substance that affects a system as critical as the brain's dopaminergic system is destined to have adverse effects under some circumstances. The first adverse effects of low-dose deprenyl were discovered by long-time practitioners of life extension who had been using l-dopa for life extension purposes. L-dopa had been shown to increase life expectancy in animals by helping to replace the dopamine normally lost by aging. (L-dopa is also a potent growth hormone releaser.) The addition of low-dose deprenyl to low-dose l-dopa usually results in a rather alarming adverse reaction, often after a delay of a week or two after the combination is begun. The symptoms are usually those of a dopamine overload, including involuntary muscle movements, nausea, headache, or just plain feeling lousy.

    About five years after life extensionists discovered the results of the deprenyl/l-dopa interaction, reports in the scientific literature began to appear showing the long-term adverse effects of the deprenyl/l-dopa combination on Parkinson's patients. A study in the Dec. 16, 1995 British Medical Journal reported the worst long-term effects, with a sharp increase in deaths occurring in the period between 29 months and 41 months after beginning the deprenyl/l-dopa combination, with an overall death rate nearly 60 percent above that of patients using l-dopa alone.

    More recent reports on the DATATOP study have shown that the early benefits of deprenyl vanished when it became necessary to add l-dopa to the patient's therapy.

    Deprenyl is known to multiply some of the effects of l-dopa. It is likely that the deprenyl/l-dopa combination produces what is effectively an l-dopa overdose. Until more is known about the deprenyl/l-dopa combination, healthy people (and probably even those with mild Parkinson's disease) should avoid the deprenyl/l-dopa combination.

    There are also possible long-term adverse effects of low-dose deprenyl without l-dopa. Deprenyl greatly increases the activity of superoxide dismutase (SOD), one of the body's natural antioxidants. On the surface, this appears to be a good thing. For years, people have been trying to find ways to increase natural antioxidant activity, especially SOD. In the process of destroying some free radicals, though, SOD, like most antioxidants, produces other free radicals. This is why complementary antioxidant systems must be kept in balance.

    This is true whether the antioxidants are vitamin supplements or the natural antioxidant systems of the body. (See Appendix A: Notes on Antioxidants)

    This is one reason why it is wise to avoid whatever is the latest antioxidant fad. Just because a substance is an antioxidant doesn't mean it is good for you.

    One of several causes of impaired mental development in Down's syndrome is the excess production of SOD. The excess SOD destroys some types of free radicals but, in the process, SOD produces more of the dangerous hydroxyl radical than the other antioxidant systems can handle. There is a possibility that deprenyl, even at low doses, could produce some of the same kind of damage.

    Obviously, if we could re-balance the antioxidant systems, the excess SOD activity could be turned from a problem to an advantage. It will be years before we know the effect of long-term low-dose deprenyl in humans. In the meantime, here are some things for anyone contemplating low-dose deprenyl for life extension to consider:

    • The three major natural antioxidant systems in the body are SOD, catalase, and glutathione peroxidase. Deprenyl raises SOD activity markedly, catalase activity slightly, and glutathione peroxidase activity none at all. The nutritional supplement N-Acetyl-Cysteine (NAC) raises glutathione peroxidase levels and should help to re-balance the body's natural antioxidant system in those using deprenyl.
    • It is probably unwise for anyone to use deprenyl without rather large doses of supplementary antioxidant vitamins, especially vitamins C and E.
    • The increased SOD activity induced by deprenyl is greater in females than males. The lifespan studies with deprenyl that produced positive results in animals were always done with male animals. Female animals did not have positive results in lifespan studies. The ideal dose of deprenyl in women appears to be less than the ideal dose for men. The use of supplementary antioxidants in women taking deprenyl is correspondingly more important than for men taking deprenyl.


    Dosage for Life Extension.



    The optimum dose of deprenyl for life extension purposes is unknown. Extrapolation from animal experiments would indicate that it is about 5 mg. every other day. Some scientists, though, have suggested that people in their forties begin with 5 mg. per week and gradually increase to about 5 mg. per day by the time they reach their seventies. Another complicating factor is that the early deprenyl experiments were done only with male animals. A recent study using male and female rats indicated that the optimal dose for females is much smaller than the optimal dose for males. Until more research is done, it may be prudent for healthy women under 70 to limit their dosage to 5 mg. per week.

    The half-life of deprenyl in the body is only a few hours; but once it enters the brain, its effects are very long-lasting. The half-life of MAO-B inhibition in humans has been measured to be about 40 days, therefore, deprenyl probably need not be taken daily by persons who do not have a neurological disease.

    The successful life extension experiments with deprenyl have been done in rodents, where the half-life of MAO-B inhibition due to deprenyl is 8 to 11 days, as opposed to 40 days in humans. This indicates that life extension doses extrapolated from rat studies may be 4 to 5 times too high for humans.

    (In the study where the half-life of MAO-B inhibition was measured in humans, it was about 38 days in the normal subjects and 43 days for patients with early Parkinson's disease. The normal subjects were four non-smoking males ages 62 to 69. The Parkinson's patients were 2 males and 2 females ages 62-70.)



    [A personal note: After all this, sometimes confusing, information about deprenyl, you are probably wondering how much, if any, deprenyl I take. In 1989, I began taking 15 mg. a week. I soon reduced the dosage to 5 to 10 mg. of deprenyl weekly. In 1996, I reduced my dose to 5 mg. a week and started using liquid deprenyl citrate. Since the most well-tested brand of liquid deprenyl citrate became unavailable, I began using 5 mg. per week in the tablet form. I also take 1000 mg. of NAC per day and at least 400 I.U. of vitamin E and 1000 mg. of vitamin C per day. I also take a lot of lipoic acid.]

    (I should also note that some of the information in this chapter may be somewhat out-of-date since the last really major update to this particular chapter was made in 1999. In the subsequent ten years, I only made some minor changes until 2009, when another significant update was made.)

    In the United States, the FDA has approved a deprenyl skin patch that is sold under the brand name Emsam for depression. It is not known whether this might be a better form of deprenyl for preventive and life extension purposes. Since the skin patch releases deprenyl at a steady rate over a 24 hour period, there is some reason to believe that using the 6 mg. Emsam patch every other day might be a good approach to the preventive medicine uses of deprenyl. The big downside, for now, to the use of Emsam is its extremely high price tag. As of this writing, the price of Emsam is more than 500 dollars for a box of 30 of the 6 mg. patches. This is more than 30 times the price of deprenyl tablets.)



    About Joseph Knoll



    Dr. Knoll began taking deprenyl in 1988, more than two decades after he developed it. Dr. Knoll is Past Chairman of the Department of Pharmacology and Professor Emeritus at Semmelweis University. He has developed many other new medicines in addition to deprenyl and has published 852 scientific papers and holds 53 patents.

    Dr. Knoll is a survivor of the Nazi concentration camps during World War II. According to an interview with Knoll in the December, 1992 issue of Longevity magazine, his weight was down to just 81 pounds during one point in his captivity. He was in a group of 65 people, of which only two survived.

    "The body is a servant to the brain," according to Knoll. This belief has driven much of his professional life.
     
    Last edited: Feb 8, 2017
  6. JanSz

    JanSz Gold

    By Alex Fergus.
    On Fatty Acids.
    [​IMG]
    His thread:
    My Quest To Be Better Than The Rest

    https://forum.jackkruse.com/index.p...better-than-the-rest.16795/page-4#post-201312

    his fatty acids;

    [​IMG]



    https://www.alexfergus.com/blog/pufa-s-the-worst-thing-for-your-health-that-you-eat-everyday

    on Facebook
    Quantum Health: Light, Water and Magnetism
    https://www.facebook.com/groups/872967099382852/permalink/1449734751706081/

    My comments there:

    Jan Szulc Nice article. Good advice.
    There is a lots of problems about fats that need better understanding, at least by me.
    I have a problems with some general statements that are expressed often, parts of them are likely true the other part not so.
    One, Americans consume a lot of omega6, I think it is likely true.
    The other, omega3 fats, fish oil, seafood suppress omega6, true.
    The further discussion always implies that most people float in omega6, I do not think that is true.
    One likely possibility,
    fish oils salesmen won the war.
    That eventually includes seafood.

    May way of dealing with this is to do blood test for fatty acids. Then read results.
    One problem, fish oil salesman thought of that already.
    There are numerous testing laboratories that do (possibly good testing) but present results in such a way that protect them from lawyers but still recommends eating fish oil.
    Not all fatty acids tests are equal.
    Knowing proper results of fatty acids analysis is just one step in right direction.
    The other one is how to read the results.
    Remember, even small excess of O3 will suppress O6, those fats are not of equal power. One fights with knife the other with gun.
    On the forum number of people posted their analysis.
    Most have excess of O3. Often EPA is high, but also quite often the beloved DHA is high.

    The other tidbit.
    Most people that are sick of neolithic diseases have large excess of omega3.
    Fibromyalgia, Adrenal fatigue, Multiple sclerosis, Parkinson's, Autism, ALS come to mind.
    Excessive Omega3 may be major reason why they are sick.

    Looking at fatty acids analysis,
    some people have (generally) high fatty acids, ie, almost all types are too high.
    The reverse, some are low across the board. Usually cholesterol is also low.
    What makes them that way? What to do to bring it back to normal. To me, this is my most important question.


    Important topic. Thanks for bringing it in.
     
    Last edited: Feb 9, 2017
  7. JanSz

    JanSz Gold

    upload_2017-2-9_9-51-22.png

    ......................................................
     
  8. JanSz

    JanSz Gold

    https://forum.jackkruse.com/index.php?threads/auotimmune-reversal.19055/#post-208261
    Auotimmune reversal?
    -------------------------------------------------
    [qu ote="prime1, post: 208239, member: 19252"]I haven't read that but just finished up lecture on eating carbs in the right season. And, wheat is good for you in the right season. More than a 3 hour lecture and my head was full. The oringal paleo diet had carbs in it and in fact more gluten then what we consume today. Shoots the whole gluten free crap out the window.

    Dr Douillard -

    http://eatwheat.lifespa.com/

    Dr Douillard completely crushes Dr Perlmutter - Great presentation:



    =====================================================================
    [qu ote="JanSz, post: 208258, member: 933"]

    Published on Jan 24, 2017
    Dr. John and Dr. Mercola Explore the Benefits of Wheat

    Watch the podcast: http://lifespa.com/episode-42-dr-john...

    ---
    1960, sugar industry paid Harward Research to blame heart disease on cholesterol and not on sugar.
    time 14:50

    ---
    time 25:40 how to repair gut
    ---

    Ground-Breaking News Regarding a Gluten-Free Diet...

    Join Dr. Joe Mercola and Dr. John Douillard as they discuss the newest research and information about the changing posture regarding being gluten-free. The 16-billion-dollar-a-year gluten-free industry has tainted the truth about wheat and is replacing organic, whole grain wheat with more "processed foods" like gluten-free bread – which is the most common cause of gluten intolerance!

    Don’t miss this exciting interview where Dr. Joe Mercola, author of The No-Grain Diet, revises his position on wheat. The gluten-free pendulum is shifting, and this interview is a MUST-SEE![/quote]
    =======================================================

    Processed flour ---->high GI
    Whole grain flour---->lower GI (wonder how much lower??)

    yes,
    https://www.amazon.com/Hodgson-Mill-Whole-Grain-5-Pounds/dp/B004IN601I/ref=sr_1_2_a_it?ie=UTF8&qid=1486600835&sr=8-2&keywords=whole rye flour

    [​IMG]

    Rye Sourdough Starter
    Made the Breadtopia recipe for Artisan Sourdough Rye bread (Swedish Rye Bread)
    [​IMG]


    https://www.amazon.com/Cuisinart-CB...d-Maker/dp/B0009VELTQ/ref=dp_ob_title_kitchen

    Cuisinart CBK-200 2-Lb Convection Bread Maker
    [​IMG]

    This machines assume that yeast will be used. (I think)
    Wonder how they would work with sourdough starter?

    ======================================================================

    https://www.quora.com/What-is-the-difference-between-whole-wheat-flour-and-refined-wheat-flour

    What is the difference between whole wheat flour and refined wheat flour?
    And why is refined wheat flour unhealthy?

    Wheat flour = atta, refined flour = maida.

    Wheat has 3 components: germ,endosperm,outer layer

    Whole wheat flour has all.

    Maida has only endosperm.

    Atta has fibre, maida doesn't.

    Due to fibre content, atta is processed slowly n for long time in our body.

    Maida is certainly not good, if one has diabetes it should be completely avoided.
    =============
    Maida is basically endosperm of wheat grain whereas wheat flour or atta contains husk bran, germ, and endosperm of wheat.

    ========================[/quote]
     
    Last edited: Feb 11, 2017
  9. JanSz

    JanSz Gold

    Diet and Seasonal Eating
    In my book, The 3-Season Diet, I discuss how nature has 3 growing seasons. The spring, summer and fall are the growing seasons, with winter as nature’s dormant season. In this section, learn how to eat with the seasons, according to your body type and the natural digestive cycles, and the importance of eating regular meals in a relaxed way.

    http://lifespa.com/category/diet-seasonal-eating/

    5 Required Foods for Winter Wellness
    http://lifespa.com/5-required-foods-winter-wellness/
     
  10. JanSz

    JanSz Gold



    Dr. John and Dr. Mercola Explore the Benefits of Wheat

    time 25:00
    ginger
    cumin
    coriander
    fennel
    cardamon

    they re-boot production of hcl, pancreatic enzymes, bile

    turmeric
    black pepper


    black cumin (more potent, rough, use light)
    black sesame seeds

    beets,
    celery
    apples

    improve bile, bile movement
    artichoke
    cardamon
    fenugreek


    time 37:00 lymphatic system, make it move, (gut and brain)
    berries, cherries, mulberries, cranberies,
    to move,
    brain lymphs drain when we sleep
    movement and re-bounding
    nasal breathing
    runners high
    when you have to breathe thru mouth=too much exercise=more harm than good
    breathing thru mouth tells body that there is emergency=strees
    constant stress is killer (including micro-biome) 45:30

    Hanging upside down is not good
    standing on a head is good

    photo-biology
    sun salutation, facing sun
    http://www.wikihow.com/Do-a-Sun-Salutation

    SAD light are dangerous (just blue), natural morning sunlight is great

    wheat belly--bad description, should be sugar belly
    it is about glycemic index
    refined wheat-->high GI
    whole wheat--->much lower GI

    have all six tastes at each meal
    sweet, sour, salt, pungent, bitter, astringent
    meal become emotionally stable and balanced, no need for desert
    reset fat balance as primary fuel by (meals far apart, no snacks in between, chance for insulin to fall down enough)

    in his book is description on how to make bread
    some breads can be fermented long enough to have it gluten free, microbes digest all sugar

    ---------------------------------------------------------
    http://www.bonappetit.com/test-kitc...d-breads-for-the-gluten-sensitive-taste-great

    Long-Fermented Breads for the Gluten-Sensitive

    "Most of the plastic-wrap bread you find at grocery stores is made very quickly with yeast--it goes from flour to plastic-wrap in three hours or less," says Stephen Jones, a wheat breeder who is the director of the Washington State University Research Center at Mount Vernon.

    What that means is that gluten proteins don't have time to break down,
    as they would in a bread made by
    traditional methods, where fermentation takes place over 18 to 25 hours,
    and that makes it harder to digest.
    It's possible, celiac experts theorize, that after years of eating highly processed bread, our guts are rejecting it.
    ---------------------------------------------------------


    https://www.mylife.com/joseph-mercola/josephmercola
    Joseph Mercola
    Ormond Beach, FL
    Male, Age 62, 07/08/1954

    http://www.city-data.com/city/Ormond-Beach-Florida.html
    Population in 2014: 39,075 (97% urban, 3% rural). Population change since 2000: +7.6%

    Zip codes: 32176.

    32176
    262 homes for sale.
    http://www.zillow.com/homes/for_sal...1,-80.894738,29.161755,-81.374016_rect/10_zm/

    ............................
     
    Last edited: Feb 11, 2017
  11. JanSz

    JanSz Gold

    http://lifespa.com/3seasondietchall...7fef09c7143c5bdd6b72a38ffc18c70a10f5bb60a184d

    In the winter, we naturally crave soups, nuts, warm grains, and other high fat and protein foods such as meat and fish.

    In the spring, we want salads, berries, and leafy greens — a naturally low-fat diet.

    And in the summer, when the days are long and hot, we require cooling and high-energy foods such as fruits and vegetables, which are a naturally available high carbohydrate diet.
     
  12. JanSz

    JanSz Gold

  13. JanSz

    JanSz Gold

  14. JanSz

    JanSz Gold



    Published on Feb 5, 2017

    Modern Science Returns to Ancient Grains | John Douillard's LifeSpa

    In 1986, Bob’s company Montana Flour & Grains introduced the natural food industry to an ancient Egyptian wheat, called khorasan (similar to durum wheat). This grain was marketed under his own brand name, KAMUT®, (the ancient Egyptian word for wheat). Through the trademark, Bob has been able to preserve an ancient grain and guarantee it has not been genetically modified or altered. KAMUT® Brand khorasan wheat is grown under strict production guidelines and is exclusively grown organically.
    ==============================================================

    https://www.amazon.com/Bobs-Red-Mill-Organic-Kamut/dp/B004VLVM9M/ref=sr_1_1_a_it?ie=UTF8&qid=1487028063&sr=8-1&keywords=Organic Kamut Flour

    [​IMG]
    Bob's Red Mill Organic Kamut Flour, 20 Ounce
     
    Last edited: Feb 14, 2017
  15. Jack Kruse

    Jack Kruse Administrator

    name of the thread is low cortisol level: This thread has really gone off the rails.

    What does full spectrum sunlight destroy naturally? UV light lowers adrenalin the stress hormone of the sympathetic nervous system from the PVN; while the sun's photons have the ability to re zip collagen that cortisol release at 4AM began to unwind to wake our body and mind up at dawn from sleep. When we are missing UV and/or full spectrum of sunlight in someone who is chronically stressed, sleep cannot be induced and cortisol is flatlined.
     
  16. JanSz

    JanSz Gold

    upload_2017-2-14_11-11-1.png

    http://study.com/academy/lesson/mitochondrial-christae-definition-function-quiz.html

    ------------------

    https://www.jackkruse.com/reality-7-blood-chlorophyll-types-food/#comment-430344
    REALITY #7: BLOOD AND CHLOROPHYLL TYPES AND FOOD

    Metallic hydrogen (H+) = the energy plasmoid of life.
    H+ = a room temperature superconductor that life uses to make life possible


    .[​IMG]


    The more connected you are to the sun and Earth’s magntic flux the less one needs electrons from food. It is a simple quantum rule. The more continous the connection the more electric charge cells can hold which can be used as an electromagnet vice grip to compress and squeeze hydrogen to changes its physics. It takes a tremendous amount of energy from the environment to make H+. The sun’s photoelectric power and the Earth’s magnetic flux can provide that electric power. Moreover, if you convert it back to molecular hydrogen (H2), all that energy is released naturally to the system built around it.
     
    Last edited: Feb 14, 2017
  17. JanSz

    JanSz Gold

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    JanSz Gold

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    JanSz Gold

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    JanSz Gold

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