Yes, that link is a life lived without enough sun.

If the deficit is mild to moderate you get diabetes.

If the deficit is severe, your heteroplasmy rate raises and you wind up with pancreatic cancer.

RUMINATE ON IT


Vitamin D reduces diabetes risk AND pancreatic cancer risks are linked!

The knee jerk reaction is to think that you just need to take 2,000 IU of D3 and you should be good.

BUT, consider this...

We make Vitamin D from sunlight exposure. God/evolution would not have linked its production to Sun exposure if we were supposed to avoid the sun and wear sunscreen when we were outside.

What if it’s more than just Vitamin D that is responsible for this disease control? What if it was the actual sunlight that changed the biophysics and allowed our bodies to be healthy and avoid disease?

In fact, this is the case. Exposure to full-spectrum sunlight and avoiding overexposure to blue light is critical to staying healthy. Clearly, overexposure to the Sun and getting burned is not a good thing.


https://www.ncbi.nlm.nih.gov/m/pubmed/27030935/

 

What we know is that pancreatic tumors are wired for high-glucose metabolism. We also know that Infrared photobiomodulation (PBM) therapy improves glucose metabolism. The question is will glucose rich pancreatic tumors respond to IR therapy? This has a high probability; IR light not only penetrates deep into tissue, but hemoglobin stores up light for redistribution.

 

During the late 1970's, the Soviet Union employed intravenous laser therapy (IVL) by introducing UV light directly in the blood system. It seemed to activate vitamin D3 production greater than sunlight; however, we need to take into account the latitude of the Russian scientists. The therapy was used mostly with CVD patients. The question is - If a strong source of full-spectrum sunlight is not a available (wrong location, time of year or season), can IVL therapy be considered as part of the treatment?

 

From a clinical standpoint, following procedure should prove interesting: inject the tumors with an UV attractant compound in a glucose solution using ultrasound guidance. Once the tumors have uptaken the compound, use UV laser fiberoptics to inject light into the tumors. If the light intensity great enough, the tumors will explode. Depending on the size of the tumors, asperation maybe required to remove the material.

 

Diabetes acute onset is always linked to seasons with suboptimal sunlight. No shocker to any Black Swan. Adams, S. F. (1926). The seasonal variation in the onset of acute diabetes: The age and sex factors in 1,000 diabetic patients. Archives of Internal Medicine, 37(6), 861-864. https://doi.org/10.1001/archinte.1926.00120240133010

 

In my thesis – an argument for high cholesterol https://forum.jackkruse.com/index.php?threads/granpa-johns-optimal-journal.23952/page-5#post-310676
I investigate the cholesterol molecule. It turns out it has hypoxic superpowers -> hypoglycemia.

Most researchers don't even try to formulate the mechanism of action. At best author(s) will append "more research is needed."
But I believe you know why... I think I do.

The question is always - What is the therapeutic dose?
In this "inquiry", how much hypoxia with how much cholesterol?

We know breath work comes in many flavors. The question maybe which provides the most sustained levels of lowered oxygen blood levels?
It turns out that https://forum.jackkruse.com/index.p...t-a-disease-damn-it.25242/page-19#post-307736 just maybe the ticket.

Your thoughts are invited