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IS ADRENAL FATIGUE JUST ANOTHER MANIFESTATION OF THE BLUE LIGHT HAZARD?

Discussion in 'Educating Doctors' started by Jack Kruse, Oct 4, 2019.

  1. Jack Kruse

    Jack Kruse Administrator

  2. Stephen W

    Stephen W Silver

    So even though blue light increases cortisol, and with adrenal fatigue patients having low cortisol, is this suggesting too much blue light has flogged out our adrenals hence they can’t keep producing?

    or can blue light help people with adrenal fatigue increase cortisol?..
     
  3. Jack Kruse

    Jack Kruse Administrator

    Serotonin is a protein made from an aromatic amino acid and it must be programmed by full-spectrum UV sunlight. When you are in full-spectrum artificial light you deplete your body and brain of serotonin. Serotonin is made in the eyes, skin, and gut in humans.
    The expression of serotonin (5-HT) in the retina was first reported in the 1960s. The detection of vesicular monoamine transporter and serotonin receptors in several retinal cells confirm that 5-HT is playing a neuromodulatory role in this structure. Whereas signaling pathways activated by 5-HT receptor binding have been poorly investigated so far, numerous data demonstrated that 5-HT is involved in retinal physiology, retinal physiopathology, and photoreceptor survival.
    The eye is a remarkable photosensor, can detect a single photon and transmit its signal to the brain. The receptors for light in the vertebrate eye are the visual, photoreceptor, cells of the retina. Each visual cell comprises two principal parts: the outer segment, which contains light-absorbing visual pigments, and the inner segment, which contains the nucleus, mitochondria and other subcellular organelles and which metabolically supports the functions of the outer segment. The segments are connected by the ciliary process or cilium. The inner segments of visual cells have terminals that synapse with horizontal cells and bipolar cells. The bipolar cells, in turn, form synapses with ganglion and amacrine cells. All of these cells have the enzymes that create serotonin which is tryptophan hydroxylase.
    It turns out serotonin is important for keeping melanopsin damage in the retina and arteries from occurring. I believe this is one of the key undiagnosed things going on in C-19 that few people realize that lead to clinical deterioration.
    What happens as a result? You become unable to handle acute stresses. This is why EHS, chronic fatigue, infertility, poor libido, and poor sleep all are related to melanopsin damage caused to serotonin photoreceptors. The absence of serotonin in the brain alters acute stress responsiveness by interfering with the genomic function of the glucocorticoid receptors. When these receptors are altered they no longer work as they should. When this process begins in the eye and ends in the brainstem humans get a condition called adrenal fatigue where it appears there is a global dysfunction of how their hormone receptors operate. This paper lays out the physiologic basis of this process that most allopathic doctors deny or are ignorant of the mechanisms due to a lack of knowledge about how light sculpts life. Become a member and learn more about this mechanism to stay well. https://www.frontiersin.org/articles/10.3389/fncel.2020.00128/full
     
    JanSz likes this.

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