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How does nnEMF/Blue light lower your cognition?

Discussion in 'Redox Rx' started by Jack Kruse, Jul 7, 2022.

  1. Jack Kruse

    Jack Kruse Administrator

    JanSz likes this.
  2. JanSz

    JanSz Gold

    What is the volume of new MATRIX water produced daily within human body?
    Do you agree that it is about equal to the volume of blood going thru the heart?
    Less at rest more when moving around.
    ........
    The weight of daily produced ATP is about equal to a body weight.
    ....
     
  3. Brain active transmembrane water cycling measured by MR is associated with neuronal activity

    upload_2022-7-7_21-32-20.png

    What is missing from this analysis is magnesium hydrate.
    So why does elevation of brain magnesium hydrate prevents synaptic loss?
    https://molecularbrain.biomedcentral.com/articles/10.1186/s13041-014-0065-y

    upload_2022-7-8_7-11-30.png

    (G) Magnesium ion concentration in the plasma ([Mg2+]plasma, mM) of WT (n = 14), Tg (n = 9) and Tg + MgT mice (n = 8) as measured by the calmagite method. ANOVA difference among groups (p < 0.01). (H) The intracellular free Mg2+ concentration in the red blood cell ([Mg2+]RBC) of WT (n = 12), Tg (n = 11) and Tg + MgT mice (n = 11) as measured by the flow cytometry method (fluorescent optical density, OD). ANOVA difference among groups (p < 0.05). ANOVA was followed by Bonfferoni's post hoc test. (I) Brain total magnesium content (Mgbrain, mg/g), in Tg mice (23 months old), significantly correlated with the recognition index in the short-term recognition memory test.​

    Is it possible the N-methyl-D-aspartate receptors (NMDARs) are excitatory?
    https://molecularbrain.biomedcentral.com/articles/10.1186/s13041-020-0563-z

    upload_2022-7-8_7-17-4.png

    Tripartite signalling of the NMDAR. A hypothesized model by which the NMDAR transduces signals in three parallel streams. The binding of glycine and glutamate to the ABD mediate channel gating and ionotropic function causing depolarization through monovalent cation flux and through calcium influx to downstream calcium-dependent pathways. The NMDAR can also signal non-ionotropically, through either glycine or glutamate binding independent of binding of the other co-agonist, initiating conformational changes propagated across the plasma membrane, and downstream protein-protein interactions.
    Is it possible our melanosomes have absorbed "too much" nnEMF; then thus generating and discharging "too much" non-native-electric-circuit-frequencies into the cell?
     
    Last edited: Jul 8, 2022

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