1. Registering for the Forum

    We require a human profile pic upon registration on this forum.

    After registration is submitted, you will receive a confirmation email, which should contain a link to confirm your intent to register for the forum. At this point, you will not yet be registered on the forum.

    Our Support staff will manually approve your account within 24 hours, and you will get a notification. This is to prevent the many spam account signups which we receive on a daily basis.

    If you have any problems completing this registration, please email support@jackkruse.com and we will assist you.

Elemental Diet

Discussion in 'Feedback/Suggestions' started by Tfreeman, Mar 28, 2020.

  1. Tfreeman

    Tfreeman New Member

    Has anyone ever tried an elemental diet? I have really poor digestive symptoms (burping, hiccups, gas, microbial issues) that haven't gotten better with the leptin reset. My doctor recommended doing an elemental diet for 2 weeks to give my digestion a rest and focus on working on my environment. Here are the 2 she suggested:

    https://data.integrativepro.com/product-literature/info/physicians-elemental-diet.pdf
    and
    https://store.drruscio.com/products/elemental-heal

    Anyone have any experience with this? Thoughts?
     
  2. Cuffy

    Cuffy Gold

    I've never heard of it, but I am highly skeptical about meal replacements.

    You might want to discuss with your PCP adding an ox bile supplement and/or digestive enzymes. An excellent, affordableone is NOW Foods Super Enzymes. No affiliation. I take 2-4 capsules (they recently reformulated to tablets) with each protein meal, and it keeps poop at a 4 on the Bristol scale.
     
    Tfreeman likes this.
  3. Tfreeman

    Tfreeman New Member

    I'm skeptical as well. Especially because I'm definitely still leptin resistant. I have a great brand of digestive enzymes that do nothing for me, maybe I should add some ox bile. Do you have a brand of ox bile on its own that you like?
     
  4. Sue-UK

    Sue-UK New Member

    You may find that over time, your thiamine hack improves your digestive symptoms.

    Is Thiamine Deficiency Destroying Your Digestive Health? Why B1 Is ESSENTIAL For Gut Function.
     
    Josie Thomson and Victoria B. like this.
  5. Cuffy

    Cuffy Gold

    For just ox bile, this one is good https://www.amazon.com/gp/product/B002GOFFWG/

    However, I prefer the added digestive support of betaine HCl, pancreatin, pepsin, and bromelain in NOW Supplements Super Enzyme. I've been taking these for 10 years. (I believe ox bile was added to the blend a year ago) https://www.amazon.com/gp/product/B0013OXKJA
     
    JanSz likes this.
  6. Casey55

    Casey55 New Member

    I also was following this diet. It helps to maintain the weight you have, and it is good for your stomach. I was following this diet after my buccal fat removing operation. I always wanted to get rid of my double chin, and last year, I decided to get an operation at ambrosae.com. After the surgery, I followed a two-month diet, so the fat didn't come back and create a double chin again. But if you want to lose weight, I don't think that this diet is good. I think it all depends on your body, and the best solution is to go to a nutritionist.
     
    Last edited: Sep 5, 2021
  7. JanSz

    JanSz Gold

    With very good results, I have been taking betaine HCL for ages.
    Recently found out that what I have actually been missing is (lots of) potassium.

    Thank you @DrEttinger
    ...........
     
    DrEttinger and John Schumacher like this.
  8. DrEttinger

    DrEttinger Choice, the only thing we control

    Jan,

    I love this write-up and go over it with my patients. You can see how potassium is used in HCl production. In the beginning water and CO2 are used. This is the main reason I like potassium bicarbonate. When taken you get potassium plus CO2 and water. They are all precursors in the production of HCl.

    Acetylcholine (ACh) is also needed which is dominant in a parasympathetic mode and requires adequate B1, same with ATP production. Alcohol, diabetes, oxidative stress, metabolic acidosis, and sympathetic dominance all use up B1 rapidly. I take sulbutiamine (B1) daily to support ACh and because I like my wine.

    I hope this helps.

    Dr. Ettinger
    #B1 #metabolicacidosis #ATP #HCl #Diabetes #JanSz
    https://teachmephysiology.com/gastrointestinal-system/stomach/acid-production/
     
    John Schumacher and Jason F like this.
  9. EWO

    EWO Gold

    What is your preferred brand of sublingual B1?

    Edited to add I misread what you wrote above in terms of type of b1. So far I have not been able to find a source that offers (to laypeople) what you recommend. Is there one?
     
    Last edited: Sep 2, 2021
    Jason F likes this.
  10. JanSz

    JanSz Gold

    @DrEttinger
    wonder if you have another place, link, where I could go and see the content you want us to read.
    Thank you.
    When I was trying to use the current link, I got this:
    upload_2021-9-1_20-17-18.png
     
  11. DrEttinger

    DrEttinger Choice, the only thing we control

    The stomach is a gastrointestinal organ that is responsible for preliminary digestion and destroying any potential pathogenic microorganisms that may have been ingested. It is an acidic environment with a pH that can vary between 1.5-3.5.

    This article will outline the production of stomach acid, the regulation of this and some clinical conditions that result from this process going wrong.

    Hydrochloric Acid Production
    HCl is produced by the parietal cells of the stomach. To begin with, water (H2O) and carbon dioxide (CO2) combine within the parietal cell cytoplasm to produce carbonic acid (H2CO3), which is catalysed by carbonic anhydrase. Carbonic acid then spontaneously dissociates into a hydrogen ion (H+) and a bicarbonate ion (HCO3–).

    The hydrogen ion that is formed is transported into the stomach lumen via the H+– K+ ATPase ion pump. This pump uses ATP as an energy source to exchange potassium ions into the parietal cells of the stomach with H+ ions.

    The bicarbonate ion is transported out of the cell into the blood via a transporter protein called anion exchanger which transports the bicarbonate ion out the cell in exchange for a chloride ion (Cl–). This chloride ion is then transported into the stomach lumen via a chloride channel.

    This results in both hydrogen and chloride ions being present within the stomach lumen. Their opposing charges leads to them associating with each other to form hydrochloric acid (HCl).

    [​IMG]

    Control of acid production
    At rest, the number of H+– K+ ATPases present within the parietal cell membrane is minimal. The rest are sequestered within tubulovesicles in the parietal cell. Upon stimulation the vesicles fuse with the cell membrane which leads to the increased insertion of H+– K+ ATPase into the membrane, hence allowing for the increased movement of hydrogen ions into the stomach thus increasing acid production.

    Increasing acid production
    There are three ways in which acid production is increased. The first of these is via ACh, which is released from the vagus nerve. This is released firstly during the cephalic phase of digestion, which is activated upon seeing or chewing food, leading to direct stimulation of parietal cells via the vagus nerve. It is also produced during the gastric phase of digestion when intrinsic nerves detect distension of the stomach, stimulating the production of ACh by the vagus nerve.

    The main regulation pathway involves the hormone gastrin which is secreted from G cells in the stomach. G cells are activated by the vagus nerve, gastrin related peptide and by peptides in the stomach lumen produced via protein digestion. Activation of the G cells leads to the production of gastrin which is released into the blood and travels through the blood until it reaches the parietal cells. Gastrin binds to CCK receptors on the parietal cells which also elevates calcium levels causing increased vesicular fusion.

    Finally, enterochromaffin like cells in the stomach secrete histamine which binds to H2 receptors on the parietal cells. These cells release histamine in response to the presence of gastrin and ACh. This leads to increased fusion however it is via the secondary messenger cAMP as opposed to calcium in the other methods.

    Decreasing acid production
    There are a number of ways in which acid production can be decreased.

    The first of these is via accumulation of acid in the empty stomach between meals. This increase in acid leads to a lower pH within the stomach, which inhibits the secretion of gastrin, via the production of somatostatin from D cells. Once food has been broken down into chyme, it passes into the duodenum, triggering the enterogastric reflex. This reflex can be stimulated by distention of the small bowel, if there is excess acid in the upper intestine, the presence of protein breakdown products as well as excess irritation to the mucosa. Inhibitory signals are sent to the stomach via the enteric nervous system, as well as signals to medulla – reducing vagal stimulation of the stomach. The enterogastric reflex, is important is slowing down gastric emptying when the intestines are already filled.

    The presence of chyme within the duodenum also stimulates entero-endocrine cells to release cholecystokinin and secretin, both of which play a variety of important roles in completing digestion, but also inhibit gastric acid secretion. Secretin is released by the S cells of the duodenum when there is excessive acid production in the stomach.

    Other hormones including glucose dependent insulinotropic peptide (GIP) and vasoactive intestinal polypeptide also work to decrease acid production in the stomach.
     
    Last edited: Sep 3, 2021
  12. DrEttinger

    DrEttinger Choice, the only thing we control

    Benfotiamine
     
    John Schumacher, EWO and JanSz like this.

Share This Page