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Dopamine and kids.......

Discussion in 'Optimal Kids' started by Jack Kruse, Feb 6, 2020.

  1. Jack Kruse

    Jack Kruse Administrator

    Brent Patrick likes this.
  2. Jack Kruse

    Jack Kruse Administrator

    NATURAL DOPAMINE CREATION

    Making dopamine begins with the interaction of UV light and tyrosine for humans. There is another key link also tied to UV light I mentioned in Ubi 24 blog. Bright full-spectrum outdoor light stimulates the release of the retinal transmitter dopamine, which is known to be able to block the axial growth of the eye, which can also lead to eye elongation. Bright full-spectrum light contains UVA light. When UV light is missing dopamine levels are lower in the eye. Anytime dopamine is lowered, it also changes the size and shape of the globe and the ciliary muscles in our eye that controls our lens shape. Anything that affects the lens affects the wavelengths that enter the retina. This changes the circadian entrainment mechanism and alters the amount of sleep needed to regenerate the SCN. Lack of dopamine causes an elongated eye. Barreto et al. have shown that in the presence of Fe(NO3)3 two broad bands of dopamine absorbance appear, with a minimum of 270 nm and maxima at 437 and 740 nm. 437 nm is in the blue/purple range just below melanopsin’s key frequency window. The UV range is completely missing in all artificial lights today. It is also missing IR-A light too. Shorter wavelength light (260-400nm), that is present can also penetrate into the orbit, eye, and eventually into the deep brain structures of the frontal lobes to regulate dopamine levels because of the tyrosinase enzyme. It is the rate-limiting enzyme in the creation of dopamine and melanin. Dopamine is a fundamental controller of the hormone cycle of all things alive. Tyrosinase acts upon the amino acid tyrosine. Tyrosinase is an oxidase (enzyme) that is the rate-limiting enzyme for controlling the production of pigmented proteins like dopamine and melanin. Dopamine, in the presence of tyrosinase, covalently modifies and inactivates tyrosine hydroxylase in a classic feedback loop. Tyrosine kinases are now targets of Big Pharma for cancer drugs.
     
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