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Crohn's and the IBD story in the gut.

Discussion in 'Educating Doctors' started by Jack Kruse, Jan 13, 2019.

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  1. Jack Kruse

    Jack Kruse Administrator

    Leaky lymphatics causes visceral fat hypertrophy = melanopsin dysfunction in the arteries of the gut. Guess why? What happens to any tissue or anything that loses energy in the universe? They get larger by scaling laws. What happens to your ankle when it gets sprained? What happens to your heart as it fails? what happens to a star that is dying?

    “the hypertrophy of mesenteric adipose tissue may result from mispatterned and ruptured lymphatic vessels. Alteration of mesenteric adipose tissue was associated with activated NF-κB signaling pathway.” How does NF-kappa beta link to this story? Melanopsin causes increases in this inflammatory signal by destroying photoreceptors in the gut. This begins in the RBC's of people with Crohn's.

    When will they learn it is a story of aberrant light?
    https://academic.oup.com/ibdjournal/article-abstract/25/2/283/5117900


    And then there is this.

    https://forum.jackkruse.com/index.php?threads/redox-is-linked-to-rbcs-circadian-clocks.22813/

    And the link to NRF-2

    When will the food guru's learn it is always about light?

    https://onlinelibrary.wiley.com/doi/full/10.1111/php.12995
     
  2. Jack Kruse

    Jack Kruse Administrator

  3. Jack Kruse

    Jack Kruse Administrator

    The parabiosis longevity link = astrocytes loaded with Vitamin C = Belousov effect.

    MANF (mesencephalic astrocyte-derived neurotrophic factor) is one of the factors responsible for rejuvenating the transfused older mice according to new research. Shocking no one who reads biophysics.

    We know that MANF regulates metabolism and immune response in flies, mice, humans. what they forgot is that none of them can make their own Vitamin C either.......hence why the parabiosis effect is an RBC's effect.

    The myopia of researchers amazes me.
     
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